How do you get cerebral arteriosclerosis

First, fat infiltration theory, under normal physiological conditions, lipids in the filtration of arterial intima, can then pass through the middle and adventitia of the nutrient vessels, back to the blood; and atherosclerosis is due to excessive lipid filtration of arterial intima, can not be well filtered back into the nutrient vessels, so that it is deposited in the subintima of the vascular wall. Catecholamine, kinin and other vasoactive substances, as well as smoking and other vasospasm for a long time, resulting in vascular wall hypoxia, malnutrition and permeability changes; and because of the elderly vascular wall mucopolysaccharide metabolism disorders, etc., can make lipids easily deposited on the vascular wall. Third, thrombosis theory, due to intima damage, platelet adhesion to the local and form thrombosis. In recent years, with the development of electronic medical technology, the study of the etiology and pathogenesis of cerebral arteriosclerosis has opened up a broad prospect.