Hepatic encephalopathy caused by pseudoneurotransmitters

Pseudoneurotransmitters cause hepatic encephalopathy, from the perspective of nerve impulse conduction. The conduction of nerve impulses is completed through transmitters, neurotransmitters are divided into excitatory and inhibitory transmitters. Under normal circumstances, the two maintain a normal physiological balance. Excitatory neurotransmitters include dopamine, noradrenaline, acetylcholine, glutamate, aspartate, etc. in catecholamines. This aromatic amino acid in food is normally converted by intestinal dehydrogenase into lamine and phenylethylamine, which are broken down and removed in the liver by monoamine oxidase; however, if the liver fails, this clearance is impaired and the amines can enter the brain, where they are formed by beta hydroxylase. The chemical structure of these two is similar to the normal neurotransmitter noradrenaline, so they are called pseudoneurotransmitters. Pseudoneurotransmitters are taken up by brain cells and replace normal transmitters in synapses. Neurotransmission disorders occur. Exciting impulses do not normally reach the cerebral cortex. Abnormal conditions occur. Consciousness disorders, coma, and so on.