How does TCM treat eczema?

Chinese medicine is how to treat this eczema, Chinese medicine we call eczema wet sores, wet sores Chinese medicine this etiology, pathogenesis, we generally speaking, is because of this congenital lack of talent, and then feel the wind evil, wet evil, heat evil, caused by the blocked skin. The first one is that innate endowment is insufficient. Innate endowment is actually a bit more common. It is the resistance given to you by your parents in the womb. Is it strong or weak? We say that there are some patients who are born with insufficient endowment. They are easy to feel wind evil, heat evil and dampness evil, so eczema occurs. The second point is that from the perspective of traditional Chinese medicine, we say that diet, some people like to eat these spicy stimulation, fat sweet thick taste, these spicy products, or some people like to eat these cold food, like eating a popsicle in summer, several popsicles a day, this kind of cold food, easy to damage the spleen and stomach. Spleen and stomach is our innate source, is our biochemical source. After the spleen and stomach are damaged, the spleen governs the movement of dampness, and our dampness evil is endogenous. After the dampness evil is born for a long time, it will stagnate and give rise to fever. After the heat evil is present, it will heat the skin and stagnate. Then it will appear on the skin, showing eczema. Another point is dampness and heat. After a long time, according to traditional Chinese medicine, it will consume yin blood and hurt yin. After yin is injured and yin blood is consumed, it will show a dryness of the skin, that is, blood deficiency will cause weathering wind, resulting in a wet sore on the skin. Generally speaking, acute eczema and wet sores are still damp-heat evil caused by dampness evil and heat evil. Subacute eczema and subacute wet sores are generally caused by spleen deficiency and dampness. Chronic eczema and wet sores are generally caused by blood deficiency and wind dryness. Such a etiology and pathogenesis.